PMID:4905670: Difference between revisions
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!align=left | !align=left align='left' bgcolor='#CCCCFF' |Citation | ||
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''' | '''No author listed''' (1970) Energy coupling in the lactose transport system of Escherichia coli. ''Proc. Natl. Acad. Sci. U.S.A.'' '''65''':63-9 | ||
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!align=left | !align=left align='left' bgcolor='#CCCCFF' |Abstract | ||
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A mutant (ML 308-22) was isolated from Escherichia coli ML 308, which had lost the normal capacity to accumulate lactose analogs despite an increase in the membrane carrier activity. The exit of thiomethylgalactoside was much faster than normal, accounting for the inability of the cell to maintain high intracellular concentrations of galactosides. Growth of the mutant on lactose was normal at high concentrations of sugar and impaired at low concentrations. This transport defect appeared to be limited to the lactose transport system as D-fucose and alpha-aminoisobutyric acid uptake and accumulation were normal. It is inferred from the data that the mutant possessed a defect in the coupling of metabolic energy to lactose transport. | A mutant (ML 308-22) was isolated from Escherichia coli ML 308, which had lost the normal capacity to accumulate lactose analogs despite an increase in the membrane carrier activity. The exit of thiomethylgalactoside was much faster than normal, accounting for the inability of the cell to maintain high intracellular concentrations of galactosides. Growth of the mutant on lactose was normal at high concentrations of sugar and impaired at low concentrations. This transport defect appeared to be limited to the lactose transport system as D-fucose and alpha-aminoisobutyric acid uptake and accumulation were normal. It is inferred from the data that the mutant possessed a defect in the coupling of metabolic energy to lactose transport. | ||
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[http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=4905670 PubMed] | [http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=4905670 PubMed] [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC286191 PMC286191] | ||
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!align=left | !align=left align='left' bgcolor='#CCCCFF' |Keywords | ||
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Aminoisobutyric Acids; Biological Transport, Active; Carbon Isotopes; Escherichia coli; Fucose; Genetics, Microbial; Glycosides; Lactose; Mutation | Aminoisobutyric Acids/metabolism; Biological Transport, Active; Carbon Isotopes; Escherichia coli/isolation & purification; Escherichia coli/metabolism; Fucose/metabolism; Genetics, Microbial; Glycosides/metabolism; Lactose/metabolism; Mutation | ||
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==Main Points of the Paper == | ==Main Points of the Paper == | ||
{{LitSignificance}} | {{LitSignificance}} | ||
== Materials and Methods Used == | == Materials and Methods Used == | ||
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==Phenotype Annotations== | ==Phenotype Annotations== | ||
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{| | {| border="2" cellpadding="4" cellspacing="0" style="margin: 1em 1em 1em 0; border: 1px #aaa solid; border-collapse: collapse;" id="N5223848ed4612" class=" tableEdit Phenotype_Table_2" | ||
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!| | !|Phenotype of!!Taxon Information!!Genotype Information (if known)!!Condition Information!!OMP ID!!OMP Term Name!!ECO ID!!ECO Term Name!!Notes!!Status | ||
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==Notes== | ==Notes== | ||
==References== | |||
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<references/> | <references/> | ||
[[Category:Publication]] | |||
Latest revision as of 18:16, 1 September 2013
| Citation |
No author listed (1970) Energy coupling in the lactose transport system of Escherichia coli. Proc. Natl. Acad. Sci. U.S.A. 65:63-9 |
|---|---|
| Abstract |
A mutant (ML 308-22) was isolated from Escherichia coli ML 308, which had lost the normal capacity to accumulate lactose analogs despite an increase in the membrane carrier activity. The exit of thiomethylgalactoside was much faster than normal, accounting for the inability of the cell to maintain high intracellular concentrations of galactosides. Growth of the mutant on lactose was normal at high concentrations of sugar and impaired at low concentrations. This transport defect appeared to be limited to the lactose transport system as D-fucose and alpha-aminoisobutyric acid uptake and accumulation were normal. It is inferred from the data that the mutant possessed a defect in the coupling of metabolic energy to lactose transport. |
| Links | |
| Keywords |
Aminoisobutyric Acids/metabolism; Biological Transport, Active; Carbon Isotopes; Escherichia coli/isolation & purification; Escherichia coli/metabolism; Fucose/metabolism; Genetics, Microbial; Glycosides/metabolism; Lactose/metabolism; Mutation |
| edit table |
Main Points of the Paper
Please summarize the main points of the paper.
Materials and Methods Used
Please list the materials and methods used in this paper (strains, plasmids, antibodies, etc).
Phenotype Annotations
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| Phenotype of | Taxon Information | Genotype Information (if known) | Condition Information | OMP ID | OMP Term Name | ECO ID | ECO Term Name | Notes | Status |
|---|---|---|---|---|---|---|---|---|---|
| edit table |
Notes
References
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